QUOTE : chromosomes. Normal aging happens because the chromosomes don’t regenerate all the way to their ends during the DNA replication cycle. Telomeres protect the information in those chromosomes; telomerase is the enzyme that activates the telomeres to replicate and replace themselves as they fall off. You have extra-long telomeres protecting your chromosomes, which might have already been genetic, or it might have come with the porphyria mutation when you were infected. We think that infection locked onto your DNA and has super-promoted your telomerase. So even if your telomeres were inclined to fall off as they normally would – a process that would allow the chromosomes to alter and change – your rocket-fuel telomerase makes new telomeres so fast there’s no time for apoptosis.